new deadly human-to-human-transmissible coronavirus emerges out of China
POSTED BY: 1KIKI
UPDATED: Tuesday, May 5, 2026 22:21
VIEWED: 203741
PAGE 223 of 283
But here's a link to a far more extended article with extensive quotes, and a link to her paper @ scribd.
The analysis moves MY needle more over to the 'human-made' side (though I admit I only somewhat glanced it over and didn't critically look at it for gaps, errors, or contradictions).
https://www.zerohedge.com/medical/rogue-chinese-virologist-joins-twitt
er-publishes-evidence-covid-19-created-lab
This looks to be the link to the paper https://zenodo.org/record/4028830#.X19xByXZglR
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Quote:I also forgot to mention that the population of Portland was 583,000 - and Portland metropolitan area was 2.4 million, thereby diluting any "/100,000" or per capita figures.
Originally posted by 1KIKI:
By sheer numbers, Portland doesn't come close to the 400,000+ person Sturgis superspreader event, which was followed by sharp outbreaks in both N (150/M -> 350/M) and S (100/M -> 400/M) Dakota.
The population of the entire state of North Dakota was 762,000 and South Dakota was 884,000.
Your numbers don't mean what you think they mean.
Your stated figures would amount to about 250 extra cases in SD, about 150 for ND.
Equivalent extra cases in Portland Metro would be about 100/M for 250 cases, and about 65/M for 150 cases.
Are you really claiming that Portland metro area did not increase by 100/M cases or even 65/M cases during these 100 days?
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Quote:
Originally posted by 1KIKI:
COVID-19 Can Wreck Your Heart, Even if You Haven’t Had Any Symptoms
A growing body of research is raising concerns about the cardiac consequences of the coronavirus
We’ve also known for a while that some COVID-19 patients’ hearts are taking a beating, too—but over the past few weeks, the evidence has strengthened that cardiac damage can happen even among people who have never displayed symptoms of coronavirus infection. And these frightening findings help explain why college and professional sports leagues are proceeding with special caution as they make decisions about whether or not to play.
More than a dozen athletes at Power Five conference schools have been identified as having myocardial injury following coronavirus infection, according to ESPN; two of the conferences—the Big Ten and the Pac-12—already have announced they are postponing all competitive sports until 2021. And in Major League Baseball, Boston Red Sox ace pitcher Eduardo Rodriguez told reporters that he felt “100 years old” as a result of his bout with COVID, and of MLB’s shortened season because of myocarditis—an inflammation of the heart muscle, often triggered by a virus. Said Rodriguez: “That’s [the heart is] the most important part of your body, so when you hear that … I was kind of scared a little. Now that I know what it is, it’s still scary.”
Why are these athletes (and their leagues and conferences) taking such extreme precautions? It’s because of the stakes. Though it often resolves without incident, myocarditis can lead to severe complications such as abnormal heart rhythms, chronic heart failure and even sudden death.
Here’s the background: Myocarditis appears to result from the direct infection of the virus attacking the heart, or possibly as a consequence of the inflammation triggered by the body’s overly aggressive immune response. And it is not age-specific: In The Lancet, doctors recently reported on an 11-year-old child with multisystem inflammatory syndrome (MIS-C)—a rare illness—who died of myocarditis and heart failure. At autopsy, pathologists were able to identify coronavirus particles present in the child’s cardiac tissue, helping to explain the virus’ direct involvement in her death. In fact, researchers are reporting the presence of viral protein in the actual heart muscle, of six deceased patients. Of note is the fact that these patients were documented to have died of lung failure, having had neither clinical signs of heart involvement, nor a prior history of cardiac disease.
Ossama Samuel, associate chief of cardiology at Mount Sinai Beth Israel in New York, told me about a cluster of younger adults developing myocarditis, some of them a month or so after they had recovered from COVID-19. One patient, who developed myocarditis four weeks after believing he had recovered from the virus, responded to a course of steroid treatment only to develop a recurrence in the form of pericarditis (an inflammation of the sac surrounding the heart). A second patient, in her 40s, now has reduced heart function from myocarditis, and a third—an athletic man in his 40s—is experiencing recurring and dangerous ventricular heart rhythms, necessitating that he wear a LifeVest defibrillator for protection. His MRI also demonstrates fibrosis and scarring of his heart muscle, which may be permanent, and he may ultimately require placement of a permanent defibrillator.
J.N. ... told me that COVID-19 symptoms first appeared in his case in late March. ... Just 34 years old, he was diagnosed with COVID-induced myocarditis and severe heart failure. Doctors admitted him to the intensive care unit and placed him on a lifesaving intra-aortic balloon pump due to the very poor function of his heart. He spent two weeks in the hospital, has suffered recurrences since his discharge, and now says, “I’m very careful. I’m very concerned about the length of time I’ve been feeling sick, and if these symptoms are lifelong orNOTIFY: Y | REPLY | REPLY WITH QUOTE | PERMALINK | TOP | HOME
Quote:
Originally posted by JEWELSTAITEFAN:Quote:
Originally posted by 1KIKI:
COVID-19 Can Wreck Your Heart, Even if You Haven’t Had Any Symptoms
A growing body of research is raising concerns about the cardiac consequences of the coronavirus
We’ve also known for a while that some COVID-19 patients’ hearts are taking a beating, too—but over the past few weeks, the evidence has strengthened that cardiac damage can happen even among people who have never displayed symptoms of coronavirus infection. And these frightening findings help explain why college and professional sports leagues are proceeding with special caution as they make decisions about whether or not to play.
More than a dozen athletes at Power Five conference schools have been identified as having myocardial injury following coronavirus infection, according to ESPN; two of the conferences—the Big Ten and the Pac-12—already have announced they are postponing all competitive sports until 2021. And in Major League Baseball, Boston Red Sox ace pitcher Eduardo Rodriguez told reporters that he felt “100 years old” as a result of his bout with COVID, and of MLB’s shortened season because of myocarditis—an inflammation of the heart muscle, often triggered by a virus. Said Rodriguez: “That’s [the heart is] the most important part of your body, so when you hear that … I was kind of scared a little. Now that I know what it is, it’s still scary.”
Why are these athletes (and their leagues and conferences) taking such extreme precautions? It’s because of the stakes. Though it often resolves without incident, myocarditis can lead to severe complications such as abnormal heart rhythms, chronic heart failure and even sudden death.
Here’s the background: Myocarditis appears to result from the direct infection of the virus attacking the heart, or possibly as a consequence of the inflammation triggered by the body’s overly aggressive immune response. And it is not age-specific: In The Lancet, doctors recently reported on an 11-year-old child with multisystem inflammatory syndrome (MIS-C)—a rare illness—who died of myocarditis and heart failure. At autopsy, pathologists were able to identify coronavirus particles present in the child’s cardiac tissue, helping to explain the virus’ direct involvement in her death. In fact, researchers are reporting the presence of viral protein in the actual heart muscle, of six deceased patients. Of note is the fact that these patients were documented to have died of lung failure, having had neither clinical signs of heart involvement, nor a prior history of cardiac disease.
Ossama Samuel, associate chief of cardiology at Mount Sinai Beth Israel in New York, told me about a cluster of younger adults developing myocarditis, some of them a month or so after they had recovered from COVID-19. One patient, who developed myocarditis four weeks after believing he had recovered from the virus, responded to a course of steroid treatment only to develop a recurrence in the form of pericarditis (an inflammation of the sac surrounding the heart). A second patient, in her 40s, now has reduced heart function from myocarditis, and a third—an athletic man in his 40s—is experiencing recurring and dangerous ventricular heart rhythms, necessitating that he wear a LifeVest defibrillator for protection. His MRI also demonstrates fibrosis and scarring of his heart muscle, which may be permanent, and he may ultimately require placement of a permanent defibrillator.
J.N. ... told me that COVID-19 symptoms first appeared in his case in late March. ... Just 34 years old, he was diagnosed with COVID-induced myocarditis and severe heart failure. Doctors admitted him to the intensive care unit and placed him on a lifesaving intra-aortic balloon pump due to the very poor function of his heart. He spent two weeks in the hospital, has suffered recurrences since his discharge, and now says, “I’m very careful. I’m very concerned about the length of time I’ve been feeNOTIFY: Y | REPLY | REPLY WITH QUOTE | PERMALINK | TOP | HOME
Quote:THANKS for the link, JSF! I saw that in the news and bookmarked it on my 'puter, but - as I don't for probably 95% of my bookmarks - I didn't post it here.
Originally posted by 1KIKI:Quote:
Originally posted by JEWELSTAITEFAN:
Proof that Covid was created in Wuhan Lab?
https://ca.news.yahoo.com/chinese-virologist-claims-she-proof-02570016
4.html
ETA: Despite claims that a laboratory origin has been debunked by this paper https://www.nature.com/articles/s41591-020-0820-9 , the takedown of many such papers due to poor data, faulty analysis, and unjustified conclusions, by a genuine virologist who specializes in viral evolution has left the 'origins' questions open, imo.
Apparently that doc was interviewed on Tucker Carlson a day or so after the story broke, several days sago by now.
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Quote:There IS no spike in Portland. Pendleton and Ontario do seem to have that problem, though! https://www.nytimes.com/interactive/2020/us/oregon-coronavirus-cases.h
That must be some incredible spike in cases in Portland that you are trying to deny.
tml
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Quote:WTF are you babbling about?
I had thought all reasonable folk had understood that Wuhan Virus was in SoCal before November.
Are you saying you didn't get the news, or are you denying it? Or not infusing your link with known data?
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Quote:What your poor brain seems to be missing is the absence of a SPIKE in Portland related to the protests/(riots).
By sheer numbers... during these 100 days?
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CDC abruptly removes guidance about airborne coronavirus transmission, says update 'was posted in error'
The guidance had been quietly updated on Friday, according to the CDC's website. CNN was first to report the change on Sunday. The CDC responded to CNN just before noon on Monday to say it was reverting to the previous guidance.
Despite several studies that have shown the novel coronavirus can spread through small particles in the air, the CDC page now says that Covid-19 is thought to spread mainly between people in close contact -- about 6 feet -- and "through respiratory droplets produced when an infected person coughs, sneezes or talks." This is the same language it posted months ago.
In language posted Friday and now removed, CDC said Covid-19 most commonly spread between people who are in close contact with one another, and went on to say it's known to spread "through respiratory droplets or small particles, such as those in aerosols, produced when an infected person coughs, sneezes, sings, talks or breathes."
These particles can cause infection when "inhaled into the nose, mouth, airways, and lungs," the agency said. "This is thought to be the main way the virus spreads."
"There is growing evidence that droplets and airborne particles can remain suspended in the air and be breathed in by others, and travel distances beyond 6 feet (for example, during choir practice, in restaurants, or in fitness classes)," the page said in the Friday update, which has since been removed. "In general, indoor environments without good ventilation increase this risk."
In the Friday update, the CDC had added new measures to protect yourself in others, including recommendations to use air purifiers to reduce airborne germs in indoors spaces and clear guidance to "stay at least 6 feet away from others, whenever possible." The updated CDC page had also changed language around asymptomatic transmission, shifting from saying "some people without symptoms may be able to spread the virus" to saying "people who are infected but do not show symptoms can spread the virus to others." That language has now been removed.
Also on Friday, CDC updated its coronavirus testing guidance to stress that anyone who has been in contact with an infected person should be tested for coronavirus. A controversial earlier update was not written by CDC scientists and posted online before it had undergone the normal scientific review process, two sources confirmed to CNN last week.
https://edition.cnn.com/2020/09/21/health/cdc-reverts-airborne-transmi
ssion-guidance/index.html
https://www.nytimes.com/2020/09/21/world/covid-19-coronavirus.html
https://www.latimes.com/world-nation/story/2020-09-20/coronavirus-aero
sol-airborne-spread
Also
Despite progress since July, most states are going backward with Covid-19 as doctors worry about 'a very apocalyptic fall'
https://edition.cnn.com/2020/09/21/health/us-coronavirus-monday/index.
html
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My guess is that the new guidance was based on at least 1 CDC study that showed eating at restaurants was a significant source of transmission.
https://www.npr.org/sections/coronavirus-live-updates/2020/09/11/91207
6989/adults-with-covid-19-twice-as-likely-to-have-eaten-at-restaurants-cdc-study
https://www.upi.com/Health_News/2020/09/10/Bars-restaurants-are-COVID-
19-infection-hotspots-study-confirms/6691599773114/
And, having some idea how web-pages get changed, I suspect someone at the CDC thought it was important enough to get the new guidance out sooner rather than later, even if they had to say to the Trump Admin OOPS! my butterfingers!, and even if they faced potential retaliation up to and including losing their job(s).
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